实验性高血压肾脏组织学与组织化学的变化
许龄秀,胡焕民,张伟甫
摘要(Abstract):
<正> 近代对于肾脏疾病引起血压升高(肾性高血压)的机制研究颇多。Goldblatt 氏等(1934)曾用实验方法部分钳缩狗的肾动脉,使其产生了持久性的高血压,当时他们认为引起血压的升高可能有三种机转,其基本原因在于肾脏缺血。一、肾缺血性刺激传至血管舒缩中枢,反射性的使之全身小血管
关键词(KeyWords):
基金项目(Foundation):
作者(Author):
许龄秀,胡焕民,张伟甫
DOI: 10.19367/j.cnki.1000-2707.1963.00.033
参考文献(References):
- [1] Goldblatt,H.,Lynch,J.,Hanzal,R.F.,Summerville,W.W.:Studies on experimental hypertension:Ⅰ.The production of persistent elevation of systolic blood pressure by means of renal ischemia. J Exp.Med.,59:347,March,1934.
- [2] Page,I.H.:The relationship of the extrinsic renal nerves to the origin of experimental hypertevsion,Am.J.physiol.,122:166,May,1935.
- [3] Goldblatt,H.:The renal origiin of hypertension,Physiol.Rev.,27:120,1947.
- [4] Grollman,A.:Straetural changes in the Kidneys of rats with experimental chronic hypertension,Arch.path.,43:559,1947.
- [5] Poutasse,E.F.:Occlusion of a renal artery as a cause of hypertension Circulation,13:37,1956.
- [6] Wilmer,H.A.:the correlation between the functional activity of the renal tubule and its phosphatase content,Arch.path.,37:227,1944
- [7] 刘家骝,贺萃之,陈世茂,蒋功博,姜淑芬:神经系统对于肾性高血压的影晌
- [8] Ham,h.W.:Histology,3rd Edition,P.667-669,london,1957.
- [9] Fischer,E.R.:Correlation of juxtaglomerular granulation,Pressor activity,and enzymes of macnla densain experimental hypertension.Lab.Invest.,10:707,1961.
- [10] McManus,J.F.A.:Father observation on the glomerular root of the vertebrate kidney,Quart,J.Micro.sc.,88:39,1947.
- [11] Hartroft,W.S.,Hartroft,P.M.:New approches in the study of cardiovascular disease,aldosterone,renin,hypertension and juxtaglomerular cells Federation Proceedings,20:845,1961.
- [12] 吴英恺、桂乃初:用交感神经切除术治疗原发性高血压症,中华医学杂志.37:11,1951.
- [13] 吴英恺,范度,冯传宜:原发性高血压病的交感神经切除治疗 中华医学杂志,第8号,602-604页,1953.
- [14] Goldring,W.,Chasis,H.:Itypertension and hypertensive disease,New york,1944.
文章评论(Comment):
|
||||||||||||||||||
|